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Transport and detoxification systems for transition metals, heavy metals and metalloids in eukaryotic and prokaryotic microbes.
Comparative Biochem. A — Molec. Rossman, T. Evidence that arsenite acts as a cocarcinogen in skin cancer. Schwerdtle, T. Induction of oxidative DNA damage by arsenite and its trivalent and pentavalent methylated metabolites in cultured human cells and isolated DNA.
Carcinogenesis 24 , — Shiobara, Y. Smith, A. Cancer risks from arsenic in drinking water. Styblo, M. Comparative toxicity of trivalent and pentavalent inorganic and methylated arsenicals in rat and human cells.
Tchounwou, P. Important considerations in the development of public health advisories for arsenic and arsenic-containing compounds in drinking water.
Wu, M. Association of blood arsenic levels with increased reactive oxidants and decreased antioxidant capacity in a human population of northeastern Taiwan.
Yager, J. Inhibition of poly ADP-ribose polymerase by arsenite. Yamanaka, K. The role of active arsenic species produced by metabolic reduction of dimethylarsinic acid in genotoxicity and tumorigenesis.
Yompakdee, C. A putative new membrane protein, Pho86p, in the inorganic phosphate uptake system of Saccharomyces cerevisiae. Gene , 41 — Oxford University Press is a department of the University of Oxford.
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Volume Article Contents Abstract. Article Navigation. Dopp , E. E-mail: elke. Oxford Academic. Google Scholar. Cite Cite E. Select Format Select format.
Permissions Icon Permissions. Abstract Mammals are able to convert inorganic arsenic to mono-, di-, and trimethylated metabolites.
DNA damage , arsenic , dimethylarsinous acid , monomethylarsonous acid , cellular uptake , electroporation , cytotoxicity , micronuclei.
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